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Viral fragments left behind after the immune system fights off infection may help explain some unusual aspects of COVID-19.
The lead project scientists suggest testing interferon-gamma further, alone and in combination with other treatments.
Two of 11 people who received four doses of budigalimab maintained viral suppression for a year and a half after stopping antiretrovirals.
Infants and young children who had COVID produced protective antibodies for much longer than adults.
The live-attenuated vaccine aims to produce an immune response to the entire virus rather than the frequently mutating spike protein.
Latently infected cells continue to produce viral RNA and proteins, and even defective HIV can trigger persistent immune activation.
Study findings shed light on possible causes of long COVID and could lead to more sensitive testing and personalized treatments.
Study findings identify a potential marker that could help identify people at high risk of developing long COVID.
NIH-funded research links alterations to inflammatory protein, underscores vaccine importance.
Prior research has shown that helminth infection induces both innate and adaptive immune responses.
Understanding how this variant helped people fend off the virus could lead to better ways to protect against COVID.
People with greater immune resilience may be at lower risk for severe COVID-19, AIDS and recurrent skin cancer, NIAID grantees find.
The findings provide a basis for future studies of the immune system’s role in longevity.
A group of proteins help activate cancer-fighting T cells and also trigger their exhaustion, find Dana Farber and NYU scientists.
NIH-funded study suggests need to boost CD8+ T cell response after infection.
Balyn Zaro, PhD, studies genetic differences in the innate immune system and how they can impact the effectiveness of the meds we take.
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